The Fastest Growing Disease in the World is
Raise your hand if you already knew that diabetes is the fastest-growing disease in the world.
Surprising, isn’t it? Sometimes people guess cancer, or heart disease.
It’s Type 2 diabetes.
In China, there are over 100 million people who are diabetic. In India, there are 68 million people who are diabetic.
In Mexico, diabetes is the leading cause of death, while in Australia someone is diagnosed with diabetes every 5 minutes.
All in all, one in every 4 people in a hospital bed is there because of diabetes or complications from diabetes.
But here in the US, we’re all very healthy, without many people with diabetes, right?
Of course, I’m joking.
We have just as widespread a diabetes epidemic as other countries.
How has this happened?
You already know that carbohydrates, along with too-frequent eating, are the culprit. And in this article, I’ll also explain
- What happens to your blood sugar when you eat too many carbs
- How Fat Storing Hormone resistance plays a role in pre-diabetes
- How you can ask your doctor to catch diabetes before it’s fully developed
I’m sure you’re eager to learn more so let’s dive in.
How You Develop Diabetes
When you eat too many carbohydrates, you get too much sugar in your blood. Diabetes is a disease caused by chronically high blood sugar.
Your pancreas contains cells called beta cells, whose job is to make Fat Storing Hormone. Fat Storing Hormone’s job is to get the glucose out of your blood and lower it to normal levels. Early on, even when you’re consuming high amounts of sugar and carbohydrates, your body is able to produce enough Fat Storing Hormone to keep up with the demand caused by high blood sugar.
But over a period of 10 to 20 years of chronically high blood glucose, diabetes can develop. You may not be aware that you’re in a pre-diabetic state because It takes a long time for indications of diabetes to show up on a blood test. Your pancreas is compensating and trying to help you out by producing a lot more Fat Storing Hormone than normal. In fact, it will produce 5 to 9 times more Fat Storing Hormone than someone without high blood sugar.
So, for years, you go to the doctor, get a blood test, and your blood sugar level comes back normal. You feel reassured. But in fact, in the background, your pancreas is pumping out ever-increasing amounts of Fat Storing Hormone to maintain that normal blood sugar level.
Over time, one complication of chronically high Fat Storing Hormone is that your cells will start to resist Fat Storing Hormone. This is a survival mechanism of your body, because too much Fat Storing Hormone is toxic. This results in a condition called Fat Storing Hormone resistance.
In response, your body makes even more Fat Storing Hormone, which will try to force its way into your cells and from there to the bloodstream to bring down the sugar level.
But all of this is basically silent for 10 to 20 years, and you seem healthy.
Oh, sure, you get up at night to pee, you experience sleep apnea and belly fat, and show symptoms of a fatty liver. Many of these signs point to diabetes; collectively, they’re known as metabolic syndrome.
Yet your blood glucose is still normal.
Eventually, the beta cells in your pancreas wear out. Your pancreas can no longer compensate for Fat Storing Hormone resistance. It cannot do its job holding your blood sugar to normal levels, because it's Fat Storing Hormone production is declining.
The combination of Fat Storing Hormone resistance and insufficient Fat Storing Hormone results in skyrocketing blood glucose. At that point, you’re diagnosed with diabetes.
In other words, diabetes is diagnosed very late in the chain of events leading up to it.
Yet if doctors knew more about the connection among carbs, Fat Storing Hormone, and blood sugar, they could focus on your diet earlier on in the whole chain of events, thereby preventing diabetes.
It makes sense, doesn’t it? So simple.
But why isn’t diabetes caught earlier in the process?
Let’s take a look.
The One Thing Doctors Could Do To Catch Diabetes Before It’s Fully Developed
Surprisingly, one thing doctors don’t do is check Fat Storing Hormone levels. If they did - especially in a fasting Fat Storing Hormone test called Homa IR - they would find a significant number of people with elevated Fat Storing Hormone levels but normal blood sugar levels.
Years ago, one doctor at a VA hospital tested thousands of people with normal blood glucose. A high percentage of them had high Fat Storing Hormone. He would check them up to 5 hours after a meal, and often their Fat Storing Hormone would still be high as a result of what they had eaten. So, while this knowledge was available then, it was ignored - to the detriment of the health of millions of people around the world.
Fat Storing Hormone Resistance Is Insidious
Fat Storing Hormone resistance takes place in the periphery of the body. It’s a type of what’s called peripheral resistance. This is why you may experience what’s called peripheral neuropathy, which often starts as tingling, numbness, or weakness in the toes or fingers.
You may think your foot or hand just fell asleep, when really it’s an indication of the ravages of Fat Storing Hormone resistance on your body.
Do This Next Time You See Your Doctor
Next time you’re at your doctor’s office, I want you to ask them two questions:
- Isn’t diabetes a disease of too much sugar in the blood?
- Then why aren’t you recommending that I go on a diet that’s low sugar and low carb diet?
And see what their answers are.
What’s shocking is that these basic facts aren’t taught in medical school, which is quite ironic. You can either pray your doctor learns them on their own, or you can help educate them as you take charge of your health.
I encourage you to start right now with diabetes prevention by joining my FREE 30-Day Low-Carb, No-Cheat Challenge. You can sign up here.
I look forward to seeing you successfully complete the challenge!
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Disclaimer: Our educational content is not meant or intended for medical advice or treatment.
Editor’s Note: This post has been updated for quality and relevancy.